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Treating ITBS with ART

Prepared for World Triathlon Corporation by Dr. Brian Abelson and Active Release Techniques. Click here to return to Ironmanlive.com

 

Treating
Iliotibial Band Syndrome
with Active Release Technique

Copyright by Dr. Brian Abelson DC.Calgary, Alberta, Canada
http://www.drabelson.com/
  http://www.activerelease.ca/


In this article

Treating Iliotibial Band Syndrome with ART

Anatomy and the Biomechanics of ITBS

Running and ITBS

Cycling and ITBS

Always Consider the Kinetic Chain

The Injury Process

The Cumulative Injury Cycle

The Need for a Specific Diagnosis

ART and the Treatment of ITBS

Post Treatment Exercises

Finding an ART Practitioner

 

Iliotibial Tibial Band Syndrome (ITBS) is a common injury that affects triatheletes, runners and cyclists.   Using conventional treatments, this condition never completely resolves since these treatments typically do not address all of the key structures involved in the injury.

ITBS presents as:

*   A sharp or burning pain on the lateral aspect of the knee.

*   Pain radiating up the side of the hip or thigh.

 

Vancouver International Marathon

ITBS is an overuse injury caused by the repetitive action of the iliotibial band as it moves across the lateral femoral epicondyle.

The primary functions of the Iliotibial Band are to:

*  Provide static stability to the lateral (outer) aspect of the knee.

*   Control adduction (inward motion) and deceleration of the thigh.

During a run, the ITB performs this function about 90 times per minute, or 22,000 times during a four-hour marathon.

ITBS syndrome in runners and triatheletes usually starts with excessive internal rotation of the femur.   In the triathelete, this can result from cycling, followed immediately by running.  To effectively resolve this condition, both the internal and external hip rotators must be corrected.

 

Anatomy and the Biomechanics of ITBS

The Iliotibial Band (ITB) is a wide, flat, ligamentous structure that originates at the iliac crest and inserts on the lateral aspect of the tibia, just below the knee. The iliotibial band serves as a ligamentous connection between the femur (lateral femoral epicondyle) and the lateral tibia (Gerdys Tubercle).

The ITB is not attached to bone as it passes between the femur and the tibia. This allows the ITB to move forward and backward with knee flexion and extension.

When the ITB is shortened or stressed, the repetitive motion of the knee causes friction and inflammation of the iliotibial band.

*   When the knee is flexed at an angle greater than 30 degrees, the iliotibial band moves back behind the lateral femoral epicondyle.

*   During knee extension, the ITB shifts forward in front of the lateral femoral epicondyle.

With ITBS, the bursa often become inflamed, causing a clicking sensation as the knee flexes and extends.

Cycling and ITBS

Common causes of ITBS in cyclists include:

*    Poor cleat position cause
ITBS when cleats are excessively rotated internally.
The feet should feel straight when clipped into the pedals, with no torsional or twisting stress occurring when you are pedaling.

*    Incorrect saddle height.
The saddle height should be set so that your legs are almost fully extended at the bottom of each pedal stroke. Keep the following points when adjusting saddle height:

*    Your hips should not rock back and forth when pedaling. If they do, lower your saddle.

*    If the saddle is too high, you will have to stretch your legs too far to reach the bottom of the pedal stroke.

*    Poor saddle positions.
Bike saddles that are positioned too far back cause a tightening of the ITB by forcing the cyclist to reach for the pedal with each stroke.

*    High gearing ratios.
This may cause excessive generation of force by the ITB.

*    Excessive hill work.
It is important to gradually increase hill work to avoid stressing the ITB.

Cyclist at the Penticton Ironman

 

Running and ITBS

Impingement of the ITB against the lateral epicondyle of the femur occurs just after foot strike in the gait cycle. Runners typically do not experience pain from the ITBS until the first one or two miles of a workout have been completed.[i]

Common causes of ITBS in runners include:

*  Running on slanted surfaces such as a circular track.

*  Excessive downhill runs.

*  Increasing mileage or pace too quickly.

*  Weak knee flexors and extensors.

*  Weak hip abductor causing excessive internal rotation of the knee.

*  Weak gluteus medias causing internal rotation of the thigh.

*  Pronation causing internal rotation of the knee.

*  Bow legs (Genu Varum).

*  Leg length discrepancy.

 

 

Always Consider the Kinetic Chain

The entire kinetic chain (above and below the injury) must perform properly in order to ensure effectiveness of the treatment, and to ensure optimum performance at the sport. Patterns of dysfunction will develop if any segment of the kinetic chain is not functioning properly.

Treatment can vary greatly since ITBS can be caused by dysfunctions in structures along any part of the kinetic chain. Athletes may have different soft tissue injuries, but still show exactly the same symptoms.

This is why generic treatment methodologies often do not work when treating ITBS. The following is a list of common soft tissue structures (besides the ITB) that may need to be addressed with an ITBS injury.

*   Biceps Femoris

*  Capsule

*  Collateral Ligaments

*   Gastrocnemius

*   Gluteus Medius

*   Knee Capsule

*   Meniscus

*   Patellar Tendon

*   Peroneus Longus Muscle

*   Popliteus Muscle

*   Psoas muscle

*   Vastus Lateralis

 

From Primal Pictures

 

 

 

 

 

 

 

 

 

 

 

 

 

The ITBS Injury Process

ITBS can be caused by damage to many different soft tissues, but the underlying injury process is very similar for all the soft tissues.

Changes in biomechanics causes increased stress, internal pressure, and increased friction that leads to inflammation, and eventually the formation of adhesions (scar tissue) within the ITB or along its kinetic chain.

Scar tissue is like glue, it restricts the translation or movement of adjacent tissues, causing friction, and leading to inflammation. The following diagram illustrates this classic Cumulative Injury Cycle.

Copyright Dr. Mike Leahy

Chronic irritation to the iliotibial band leads to small tears within the ITB. This produces an inflammatory reaction.

Once the inflammatory condition has started, even simple tasks such as walking can put considerable internal pressure on the ITB.

This constant internal pressure limits circulation to the tissue, resulting in decreased delivery of oxygen to soft tissues. Decreased oxygen causes several biochemical changes that result in the formation of yet more adhesions within the ITB.

This in turn creates more restrictions, inflammation, and swelling.

The body responds to inflammation by laying down additional scar tissue (cross fibers across the tissue) in an attempt to stabilize the affected area. This scar tissue:

*   Restricts motion.

*   Reduces circulation.

*   Inhibits nerve function.

*   Causes ongoing friction and pressure.

*   Results in the production of yet more cross fibers and adhesions across inflamed soft tissues.

 

 

The Need for a Specific Diagnosis

It is extremely important to be as specific as possible when identifying the soft tissue structures involved in any condition. Different athletes may present with identical pain patterns at the ITB, yet they may have completely different structures that are impairing motion or causing the injury.

Before treatment takes place, an extremely specific examination and diagnosis must be performed. It is important to look past the initial point of pain and identify all the other structures that are involved in the kinetic chain.

What to do during the acute stage of injury

Because of the inflammatory nature of this injury, it is highly recommended that the athlete use ice massage, for 3-5 minutes, over the affected area until it is numb. The more acute the condition the more essential ice massage becomes.

Usually, for conventional treatment for ITBS, a reduction or stop in training is necessary. This pause in training is often unnecessary with Active Release Technique (ART). With ART, the athlete is often able to resume training within a very short period. In fact, for most moderate cases of ITBS, the athlete is able to immediately increase the level of training after an ART treatment, without discomfort.

This is one of the great advantages of ART for improving performance. With ART, you can prevent injuries from affecting your training schedule. The equation is simple, decreased injuries means more time for training and for increasing performance.

ART and the Treatment of ITBS

To truly resolve ITBS, every structure that crosses the lateral side of the knee must be evaluated and treated. In addition to the ITB, the practitioner must also evaluate and treat the muscles, ligaments, meniscus, and capsule.

Once this is done, the hip itself must be corrected to remove the root cause of the problem.

Unfortunately, it is common for this condition to never fully resolve, since practitioners rarely evaluate and treat ALL of these structures.

With ART, resolution can be seen in over 90 percent of ITBS cases, within 4-6 visits.

Effective treatment of the ITB, or of any soft tissue injury, requires an alteration in tissue structure to break up the restrictive cross-fibre adhesions and restore normal function to the affected soft tissue areas. When executed properly, this process substantially decreases healing time, treats the root cause of the injury, and improves athletic performance.

Active Release Technique (ART®) is very successful at treating this type of injury since it removes restrictive adhesions between both the superficial and deep tissue structures along the entire kinetic chain.

The first step in treating this condition is to perform a biomechanical analysis of the athlete to determine where the restrictions are located along the entire kinetic chain.

ART treatments are specific and based upon the individual needs of each athlete. It is not a cookbook approach to treating a non-specific diagnosis.

ART finds the specific tissues that are restricted and physically works them back to their normal texture, tension, and length by using various hand positions and soft tissue manipulation methods.

Dr. Abelson treating ITBS at the Ironman Kona

 

With ART, we often see immediate improvement after we identify and work with the damaged structures.

 

 

The Value of Post Treatment Exercises

Strengthening exercises are only effective if they are executed after the adhesions within the soft tissue have been released. Attempts to strengthen muscles bound by adhesions often cause the structure to become more restricted, which in turn causes additional tension within the soft tissue.

In addition to the strengthening component, stretching, and balance work continue to be key components in correcting the problem so it does not return.

Finding an ART Practitioner

You can find a qualified ART provider by visiting the official Active Release Techniques website at www.activerelease.com. You can also call ART headquarters at 1-888-396-2727. At your next Ironman race, look for the ART Performance Care tent. Our soft tissue experts are available to help you reach your full potential.

ART Performance Care
Perform at your best!

Dr. Brian Abelson DC, ART
http://www.activerelease.ca/
http://www.drabelson.com/

Copyright: Dr. Brian Abelson 2003. All rights reserved.

Dr. Brian Abelson is Clinical Director of Edgemont Chiropractic Clinic. Dr. Abelson is a native Calgarian who graduated from Palmer College of Chiropractic West with an award for clinical excellence, holds a Level 3 Active Release Certification, and is an ART Assistant Instructor. He is also the author of the award winning websites: www.drabelson.com and www.activerelease.ca .

 

Author: Dr. Brian Abelson DC, ART

Editor: Kamali Abelson, Rowan Tree Consulting Ltd.

Edgemont Chiropractic Clinic

Bay #10, 34 Edgedale Drive N.W.

Calgary, Alberta, T3A-2R4        

 



[i] 'Iliotibial Band Friction Syndrome in Runners,' American Journal of Sports Medicine, vol. 8, pp. 232-234, 1980

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